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Dysregulated histone acetylation is increasingly linked to tumor progression and therapeutic resistance, where acetylation-driven activation of survival and redox pathways fosters drug toleranceincluding reduced sensitivity to AR-directed therapies in CRPC [25,26,27,28]

Funding We acknowledge financial support from the National Key R&D Program of China (2022YFA0912800), National Excellent Young Scientists Fund (00308054 A1045), National Natural Science Foundation of China (22178233), Talents Program of Sichuan Province, Double First-Class University Plan of Sichuan University, State Key Laboratory of Polymer Materials Engineering (sklpme 20200301), Tianfu Emei Program of Sichuan Province (2022-EC02-00073-CG), Postdoctoral Special Funding of Sichuan Province (TB2022063), Postdoctoral Funding of Sichuan University (2022SCU12099), Ministry of Education Key Laboratory of Leather Chemistry and Engineering, and National Engineering Research Center of Clean Technology in Leather Industry

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